Peter Attia· MD
those with CTP Vivi they have higher HDL levels hmm they have larger lipoprotein particle size and they have lower CTP levels that's their phenotype
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
those with CTP Vivi they have higher HDL levels hmm they have larger lipoprotein particle size and they have lower CTP levels that's their phenotype
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we actually got a very compelling data on two genotypes that seems to be functional important that are controlling lipid metabolism one of them was a ctp genotype and one it was an apoc3 genotypes and those genotypes increased from about eight to nine percent of the homozygosity in control to almost 20 in centenarians
we got a very compelling data on two genotypes that seems to be functional important that are controlling lipid metabolism one of them was a ctp genotype and one it was an apoc3 genotypes and those genotypes increased from about eight to nine percent of the homozygosity in control to almost 20 in centenarians
so the centenarians with high lp little a all have ctp they're homozygous for the ctp genotype so they have some protection to counter it
we can now add hyper functioning ctep to the list of things like hyper functioning pcsk9
ctep is a longevity Gene meaning the hypo functioning variant is as much a longevity Gene if not more than the hypofunctioning pcsk9
the centenarians with high LP little a all have CTP they're homozygous for the CTP genotype so they have some protection to counter it