Peter Attia· MD
you know when it comes to mitophagy you know these all mitochondria it's been shown in insect in rodents that this is improved with caloric restriction autoag of course all these organel in the cells
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
you know when it comes to mitophagy you know these all mitochondria it's been shown in insect in rodents that this is improved with caloric restriction autoag of course all these organel in the cells
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what do you do to have more autophagy or more mitophagy and all that I don't know I mean we know exercise does it right I mean we know that autophagy is not just fueled by chloric restriction but exercise is an incredibly potent driver of that for for similar mechanisms right you could argue that it's basically a substrate utilization problem it's the input it's the output and when you transiently deprive the output right by if you drive more output and you transiently create a deficit of energy I don't think at the cellular level the body is particularly concerned with am I short on what's coming in or am I short because too much is going out