Peter Attia· MD
Now where we could could be wrong is we don't know what is the impact of each of those pathway in humans okay most of our data is kind of animal data
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Now where we could could be wrong is we don't know what is the impact of each of those pathway in humans okay most of our data is kind of animal data
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
So there are theories, the free radical theory of aging, loss of mitochondrial function, the accumulation of senescent cells which I'm sure we'll talk about more. But for now, I think there are two things we can say that these fundamental processes almost certainly are driving tissue health. As for what actually drives lifespan, we really don't know.