The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
these all happen in metabolic dysfunction so metabolic dysfunction is leading to these modifications inflammatory response impaired glucose tolerance mitochondrial dysfunction and then what happens is the LDL starts to become inflammatory when it interacts with the vascular wall and then you have the immune response
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
there's a variety of other factors that are occurring that are leading to changes and modifications in the LDL which we can talk about and that's usually driven by metabolic dysfunction and by inflammatory signaling and oxidative stress that modify LDL particles trigger immune response and then you start to get plaque formation