The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the reason apob is going up in a high triglyceride environment is because you need more ldls to carry the same amount of cholesterol eser because so much of their carrying capacity is going towards also managing the transport of triglycerides and therefore while LDL cholesterol might remain constant it's being spread out over more particles therefore apob which is the marker of particle Concentra is going up
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
the predominant species when triglycerides goes above a certain threshold is the cholesterol poor small LLS that have decreased clearance so apob goes up
this transformation that starts to delay the lipolysis of these particles occurs somewhere at a triglyceride 100 or above