Short-term isocaloric fructose restriction in children with obesity decreased liver fat, visceral adipose tissue, de novo lipogenesis, and improved insulin kinetics.

“this study will also have important implications for questions around how quickly insulin resistance metabolic dysfunction can be reversed this is the effects of dietary fructose restriction on liver fat denova lipogenesis and insulin kinetics in children with obesity and what they did was limit processed non-food matrix contained sugar in the study short term nine days iso caloric fructose restriction meaning they did not do decreased calories in this diet decreased liver fat visceral adipose tissue de novo lipogenesis visceral adipose tissue was the adipose issue i talked about earlier within the peritoneum liver fat is a another marker of metabolic dysfunction denova lipogenesis refers to the amount of sugar specifically fructose that has converted into fat in the liver and it improved insulin kinetics in children with obesity”