The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
so there's a lot there to dig into but if you see kids who can eat anything and not get diabetic and not get fatty liver well a lot of kids are getting diabetic and fatty liver earlier but there's something going on with mitochondria and at some point this mitochondria just give up they pass out on the beach and they just start doing things badly and that could be a difference in the physiology between those ages of humans but we don't want that aldost reductase pathway
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so the mechanism is that salt increases when you eat salt the salt concentration in your blood goes up and when the salt concentration in your blood goes up it uh activates the polyol pathway it turns on this enzyme to make fructose which one the Aldos reductase yes it turns on alos reductase and that helps convert glucose to fructose
the data in humans that that um increase serum osmolality activates this pathway um or increased salt concentration in the blood can activate the Aldos reductase Pathways is very strong in humans it's very strong in all organisms