The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
how fructose when it moves into the cell through the glue 5 transporter is then phosphorylated using up phosphate groups from ATP so there's a lot of phosphate groups that are used up in the processing of fructose when it moves into a cell the ATP goes to ADP goes to a MP that's adenosine diphosphate adenosine monophosphate and then it's converted to inosine monophosphate which gets converted to uric acid
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so fructose is directly causing increased uric acid and he will also illustrate there are mechanisms by which fructose probably creates insulin resistance on its own so you're not only increasing uric acid through the production of inosine monophosphate by using up ATP to process fructose you're also creating insulin resistance which will decrease the amount of uric acid that you are excreting
the fructose metabolism will be in upregulated by uric acid so when uric acid goes up not only does it turn on the poly all pathway but it actually up regulates fructokinase as well
the fructose fructose metabolism will be in upregulated by uric acid so when uric acid goes up not not only does it turn on the polyol pathway but it actually upregulates fructokinase as well