Paul Saladino· MD
the centerpiece of metabolic dysfunction appears to be the fact that our adipocytes cannot divide that they get stuck and they overexpand
Loading…
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the centerpiece of metabolic dysfunction appears to be the fact that our adipocytes cannot divide that they get stuck and they overexpand
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
when the adipocyte can no longer do um the secreted dipakins which lead to adipocyte dysfunction it gets overly distended and that appears to be the inciting event for systemic insulin resistance or systemic metabolic dysfunction