The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
if we can expand the fat cell mass and get more fat cells that are smaller people don't get metabolic dysfunction all right this is what we saw in the sumo wrestler studies that i talked about earlier you can be fat but if you have lots of small fat cells you're not going to get inflammation you're not going to get metabolic dysfunction you're not going to get quote insulin resistance i don't like that term
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when in people who have diabetes this is well documented we see adipocyte pathology where the adipocytes the fat cells are too big if we can expand the fat cell mass and get more fat cells that are smaller people don't get metabolic dysfunction
if you have adipocyte hyperplasia which is adipocyte division you can prevent metabolic dysfunction you can be fat but metabolically healthy
big fat cells that appear to be driving metabolic dysfunction and one of the ways we get big fat cells is by eating way too much polyunsaturated fats