The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it's the liver liver can create it's old fast as well from carbohydrate which is really where the fat comes from that goes into the LDL because carbohydrates are all converted to sugars and sugars fructose and glucose primarily fructose especially is turned into fat in the liver in a process called Enuma lipogenesis
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
human studies demonstrate a rate of fractional de novo lipogenesis of 2 percent with glucose yet up to 10 percent after 6 days of high fructose feeding and it goes on to say I'm a recent human study demonstrated that fructose feeding increased fractional turnover lipogenesis to 17 percent
the mean conversion rate from fructose to glucose was 41 so almost half of the fructose we eat becomes glucose immediately and then 25 of it even more of it becomes uh becomes lactate and then a very small amount goes to de novo lipogenesis and a very small percentage ends up in triglycerides