The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when you break down polyunsaturated fatty acids or monounsaturated fatty acids they have a different ratio of fadh2 to NADH to Paul why are you telling us all this because that ratio of fadh2 to NADH 2 determines whether you're a dipa site is going to be insulin sensitive or insulin resistant
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
the the next insight that came from the protons concept is that the poly saturates refuse to trigger insulin sickening they will eventually trigger it if you put enough polyunsaturates in eventually the nadh and whatever basic level of fadh that they're producing will eventually combine together to limit calorie ingress but at that point there will still be calories coming through in excess of what the cell needs and we come back to that there will be continued insulin signaling and that will be taking the excess calories and converting it into lipid in the cytoplasm or glycogen in the cytoplasm