Peter Attia· MD
now the decision for life and death is based on the localization of cytochrome C between the mitochondria and into the cytoplasm so if it's in the mitochondria it helps you generate ATP if it's out it kills you
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
now the decision for life and death is based on the localization of cytochrome C between the mitochondria and into the cytoplasm so if it's in the mitochondria it helps you generate ATP if it's out it kills you
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if that protein gets released from mitochondria into the cytoplasm which had never been detected before it can rapidly kill a cell
as you know probably the major signal for a cell to die in anaerobic organism is for the mitochondria to release cytochrome C cytochrome C is a protein that carries the electron to cytochrome C oxidase if that machinery is not working and cytochrome C leaks out we say they mitochondrial permeability poor has been modified so that small cytochrome C leaks that's a big signal for apoptosis apoptosis or programmed cell death