The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
if beta hydraulic to ketones and sort of the root cause of why we're talking about this is that beta hydroxy butyrate can essentially oxidize Q of the Subic and if Q is oxidized it's less likely to deform a reactive intermediate called semi called superoxide anion
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
you talked about ketone molecules so you talked about beta-hydroxybutyrate in some way perhaps we understand the chemistry perhaps we don't affecting the production of the super anion radical right so super oxide and that is because of Q which is part of a complex in the mitochondria electron transport chain
through his experiences in ketogenic physiology beta-hydroxybutyrate had influence on the molecule q in the mitochondria electron transport chain and led to the production of less superoxide dismutase or excuse me less production of superoxide requiring less superoxide dismutase to manage that that radical molecule
Ketones, direct effect, yeah, on that, too. [...] Oxidize Q, makes it less available. If Q is oxidized, yeah, so. [...] If ubiquinone is oxidized, which is achieved with our beta-hydroxybutyrate metabolism, if that's oxidized, then you have less availability for that electron to react with molecular oxygen in the metabolic pathway. So you would produce less superoxide anion, which is your precursor to more reactive oxygen species.