The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
so there's input from um f d f a d transporting flavor protein all those input from complex ii in competition with input from complex one which is dealing with the nadh and they're all three of them trying to put electrons onto the coq couple that's everything has to go through the kokee pool coq couple there's no other way down the rest of the um there's no other way down the rest of the electron transport chain and it's like a bottleneck
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
now when fatty acids are oxidized if they're saturated the first step of beta oxidation generates fadh2 it can't just sit there as fadh2 because it's always protein-bound so it's um generated and transported as a flavor protein a protein with fad either fad plus embedded in it or a fad embedded in it or fadh2 embedded in it and that protein with the fad molecule it oxidized or reduced is transportable and that can go to the inner surface of the mitochondrial membrane where there is a dehydrogenase so at that point the fadh2 can donate two electrons again to coenzyme q