The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
and each time you feed complex one complex one pumps protons uh from the matrix of the mitochondrium to the uh just outside the inner mitochondrial membrane and is used to develop a almost like a hydraulic supply of protons that want to get back into the mitochondrial matrix
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
each time you feed complex one complex one pumps protons uh from the matrix of the mitochondrium to the uh just outside the inner mitochondrial membrane and is used to develop a almost like a hydraulic supply of protons that want to get back into the mitochondrial matrix but nadh the cofactor for many of the reactions at least three of them in the tca and an adh as produced as part of glycolysis eventually end up feeding complex one they don't really feed anywhere else in any bulk amount that you need to worry about so that's complex one that's one input to the ability to pump protons which is then used to generate atp using complex five but the atp synthase so complex one just deals with nadh and nothing else deals with nadh