Rhonda Patrick· PhD
But when we've looked at alcoholics, we see disturbed NAD in their liver.
Loading…
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
But when we've looked at alcoholics, we see disturbed NAD in their liver.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
So um you know the full systems analysis of it is not fully understood right and I'm not the type of person that is going to claim to know things that are that are not known. So what is generally understood which is easy to determine from gene expression patterns is that certain tissues like the liver can make NAD from tryptophan nicotenomide ribocide nicotenomide and nicotinic acid. So any precursor they can make NAD and they um there are processes in the aging liver that disturb that system. Alcoholism can disturb that system. Over nutrition can disturb that system. But it's actually a fairly robust system.