The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
if I'm deciding to be powered more by fatty since then that definitely means that instead of glucose my body is going to be trafficking more of them and therefore needing to make and circulate more of these boats the primary boat as I mentioned earlier that's going to be circulated especially an absence of eating is going to be the vldls that come from the liver and that's why LDL cholesterol going up makes perfect sense to me
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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makes sense to me because while I'm on the metabolic pathway being powered by fatty acids even if I make more ketones
by design if somebody is a fat-burning individual who is mobilizing a lot of fatty acids and putting them through the mitochondria will convert to fuel and making a lot of ketones in the process by design their LDL cholesterol will go up because the liver is making so much cholesterol at that point that it needs to package it and get it out into the circulation
of course when you are in ketosis your LDL will go up and then I love what you're illustrating here that there is evidence that LDL can be made independently of VLDL in the liver there's a paper that you note in the low carb Denver presentation that suggests that