The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
essentially the liver doesn't just convert fructose to Fat to Fat there's a bunch of different things that it can do with fructose it converts it to glucose or lactate and sends it out for the rest of the body to use it'll store its glycogen it'll oxidate it or oxidize it to produce ATP and then if all of its needs fructose are satisfied then it will convert some amount to Fat which will then be released or used later whatever it is
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
there are isotopic Tracer studies with fructose showing the fates of fructose um I think Andrew didn't do us any favors here by saying what so fructose just wants to become fat and then lustig says yes fruit fructose is the major fat making molecule and you think I don't that doesn't make any sense like number one it's very low in the blood it's a small fraction of what you're ingesting in the fructose and the amount of fat you're eating is 10 to 20 times that amount relatively speaking fructose is not going to fat that doesn't make any sense at all and when you look at these isotopic Tracer studies as you said less than 1% of the fructose actually becomes fat at the level of the liver it's converted to glucose lactate it's oxidized directly most of the fructose just gets burned yeah