The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
our hypothesis was it's the saturated fat more than the ketones because that was the other thing this guy didn't want to leave ketosis because my thought was let's just dial this back get you out of ketosis but so our hypothesis was both ketones and saturated fat can be readily converted into cholesterol
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it might be that the ketones themselves are a substrate to make more cholesterol and again we'll link to a great paper that Tom wrote on Lippa holux several years ago where he goes through the biochemistry of how saturated fats specifically and ketones could in a susceptible individual produce this phenotype