The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
in celiac disease, pathogenesis is induced by gliadin, a glycoprotein present in wheat. Ex vivo human intestinal samples from celiac patients in remission also showed zonulin released when exposed to gliadin, meaning that if you have celiac disease, even if you're in remission, you're going to get uh, zonulin released when you're exposed to gliadin.
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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meaning that if you have celiac disease even if you're in remission you're going to get zonulin release when you're exposed to gluten i believe there's also good evidence that many humans will release zonulin if not all humans when they're exposed to gluten even if they are not celiac
in celiac disease pathogenesis is induced by gliadin a glycoprotein present in wheat ex vivo human intestinal samples from celiac patients in remission also showed zonulin released when exposed to gluten
i believe there's also good evidence that many humans will release zonulin if not all humans when they're exposed to gluten even if they are not celiac