The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when you have higher Lucine it also begins to inhibit pyruvate from going into the U into the mitochondria so when you oxidize a Lucine the nitrogen that comes off of it is put onto pyruvate generating an alanine and so the body begins to recycle glucose so it becomes kind of steady state on glucose and emphasizes fat oxidation
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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Lucine is one that's referred to as a branch chain amino acid and its side chain is purely carbons it's a carbon chain that looks a lot like a fatty acid and so Lucine is one of really two amino acids that are ketogenic it is metabolized as a fatty acid and what we know is that Lucine will also activate the cpt1 enzyme the carnal pettin transfer enzyme which is the link of bringing fatty acids into the mitochondria for oxidation