Paul Saladino· MD
now what's interesting is if they give phytohemaglutinin to rats and mice those rats and mice do not thrive they lose weight they they do not do well at all
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
now what's interesting is if they give phytohemaglutinin to rats and mice those rats and mice do not thrive they lose weight they they do not do well at all
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's actually not interaction with the bacteria it's an interaction with the intestine directly these this phytohuman gluten these lectins are directly toxic to our intestines they show here phytohuman gluten and feeding has been also observed to affect small intestinal growth in a negative way
small intestinal growth caused by feeding rat uh feeding kidney bean phytohemoglobin liked into rats this is suggesting that in the small intestine you're getting growth that could lead potentially to the formation of tumors
pha which is phytohemaglutinin caused fecal protein fat and mucous glycoprotein levels to increase in germ-free animals it increased to general mucous crypt death and crypt mitotic activity and so they're saying pha stimulated growth of rats small intestine when present in the diet or instilled into the bowel lumens so it was causing inflammation and it was potentially causing issues within these rat guts when they're giving them this lectin in kidney beans
So, PHA is the lectin found in red kidney beans. They say that there are bacterial overgrowth happens in the small bowel of the rat when they give them PHA.
So, I think that the compelling hypothesis here is that these phytohemagglutinins from red kidney beans, these lectins in plants, perhaps more broadly, may damage or cause issues in the mucosal-producing cells, the goblet cells of the gut, and they may decrease the mucosal layer of the gut.
i think that the compelling hypothesis here is that these phytohim and glutamines from red kidney beans these lectins in plants perhaps more broadly may damage or cause issues in the mucosal-producing cells the goblet cells of the gut and they may decrease the mucosal layer of the gut they do appear to lead to this induced overgrowth of the small intestine and inflammation
what appeared to happen was that the mucosal surface changed when the phytohuman glutenants were administered leading to contact between intestinal bacteria and the epithelium which led to inflammation