The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
ultimately so many diseases we're dealing with now have to do with mitochondrial dysfunction and ketones are kind of an interesting alternative pathway we can use to deliver electrons to the mitochondria at a little bit different place and often bypass defects in the complexes in the mitochondrial electron transport chain
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
in neurod degenerative disorders specifically Parkinson's which is perhaps the best studied there are often defects in complex one in the mitochondrial electron transport chain and ketones are able to deliver electrons to electron transport chain in a way that sort of bypasses that complex one mutation or that complex one deficit