Peter Attia· MD
it works very well for CNS oxygen toxicity but also for things like ventolin tetras ol uh seizures pzt seizures even for absence seizures we use a
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
it works very well for CNS oxygen toxicity but also for things like ventolin tetras ol uh seizures pzt seizures even for absence seizures we use a
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there are people out there because exogenous ketones are commercially available that are using this in place of the ketogenic diet because the families are either unwilling or for various reasons unable to you the ketogenic diet and they you know use are using exogenous ketones and the feedback that I'm getting could be biased but it does seem to have an effect it obviously has an effect in animal models the animal models that we work with we give exhaustion ski tones on top of a standard rat chow which is high carbohydrate chow and it it works very well for CNS oxygen toxicity but also for things like ventolin tetras ol uh seizures pzt seizures even for absence seizures we use a particular animal model with absence seizures or absent seizure it works for that so it works for a variety of different seizure models
We know the first kind of convincing studies of this that we did was with CNS oxygen toxicity in our rat model, and in that case, the rats were eating a high-carbohydrate standard rodent chow model, and we administered via an oral route a ketone supplement in the form of a ketone ester. That's probably one of the more powerful forms of exogenous ketones that we've developed. And that had the ability to prevent CNS oxygen toxicity from happening for almost, over 500% delay in that time to CNS oxygen toxicity.