The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
so conceivably that could be having a stronger anti-inflammatory effect because you have a ketone body an endogenous metabolite that's functioning as a very powerful signaling molecule that's suppressing an inflammatory pathway
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so I think when it comes to purely a metabolic fuel I think the D beta hydroxy butyrate has some advantages
we do know that the L form gets metabolized slower so the the concentrations and the tissue may actually reach higher levels
especially the inflammation the nlrp3 inflammasome is suppressed by beta hydroxy butyrate both the D and the L form
So we say if a if a beta hydroxybutyrate supplement is recemic it has D and it has L in it
which is a form uh the mirror image of beta hydroxy of d beta hydroxybutyrate is L beta hydroxybutyrate
Uh but a DL mixture is almost like you get the benefits of the D, right? And then you get the signaling benefits of the L.
So uh beta hydroxybutyrate is produced in the body primarily uh in the form of d beta hydroxybutyrate