The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
in the setting of insulin resistance in the setting of cardiovascular disease and specifically in the setting of type 2 diabetes and in the setting of chronic kidney disease we are going to excrete less TMAO or we are going to make more TMAO and that makes that raises TMAO it's not TMAO that is causing diabetes or chronic kidney disease it is chronic kidney disease that and diabetes that are causing TMAO to rise
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
we found further that type 2 diabetes and chronic kidney disease were causally associated with higher TMAO levels our Mendelian randomization findings suggest that type 2 diabetes and kidney disease increased TMAO levels and observational evidence for cardiovascular diseases may be due to confounding or reverse causality
our mendelian randomization support that type 2 diabetes and kidney disease increase tmao levels and observational evidence for cardiovascular diseases may be due to confounding or reverse causality which is basically just fancy words for hey when you have diabetes or kidney disease your tmao goes up and that could also that your your diabetes or your kidney disease could be causing your cardiovascular disease that is called reverse causality
our Mendelian randomization findings support that type 2 diabetes and kidney disease increase TMA TMAO levels. And observational evidence for cardiovascular diseases may be due to confounding or reverse causality.
our mendelian randomization findings support that type 2 diabetes and kidney disease increase tma lev tmao levels and observational evidence for cardiovascular diseases may be due to confounding or reverse causality