The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Now the SGL2 inhibitors are approved sodium glucose co-transporter inhibitors that reabsorb glucose from the ultrafiltrate in the kidney. If you block that, then the glucose ends up in the urine. And so that's a good way to lower glucose and thereby lower insulin.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
when you block sglt2 you prevent sodium and glucose from being reabsorbed by the cell to then be put back into the plasma and therefore you will pee out more sodium and glucose