The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
there is one study that I found in the past suggesting that supplemental vitamin A vitamin A palmitate is absorbed differently in our guts than vitamin A in the retinol form which is what we find in food that is done in pigs but when they gave pigs liver and they gave pigs vitamin A palmitate they certainly saw differential absorption with the vitamin synthetic form being much more avidly and absorbed than the than the actual quote-unquote natural form
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there's also evidence from studies in pigs that the kinetics of supplements versus liver may not be the same our results indicate that intestinal metabolism contributes to the elevated of all trans retinoic acid in the systematic circular systemic circulation after supplementation with vitamin a but not after consumption with liver
if you read this paper it'll say show you that the actual supplement of liver resulted in much different kinetics in vitamin a than supplemental forms of retinal palmitate now i think that we can essentially treat retinol palmitate as retinol palmitate but it does speak to the fact that there is some difference in the way that a supplement of liver retinol palmitate is absorbed relative to actual cow's liver
women who receive the supplement rather than an equivalent dose of fried calf's liver had significantly higher concentrations of retinoids than those who did receive those who received the liver possibly because of the feud matrix the food matrix may have ameliorated the absorption rate or altered the circulating forms of vitamin a