Rhonda Patrick· PhD
But if you dampen that by like sequestering that stress with an antioxidant, we're not going to get those benefits. There are studies now showing this both in humans and mouse animals.
We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
But if you dampen that by like sequestering that stress with an antioxidant, we're not going to get those benefits. There are studies now showing this both in humans and mouse animals.
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
Also, there was another study very similar to the anti-inflammatory, where they’re saying the NSAIDs. So there’s another study that showed taking high dose, alpha-tocopherol, no, vitamin c, so antioxidants, also suppressed the insulin sensitivity effects of exercise, possibly through, because you’re not activating that whole inflammatory.
the flip side of this is that reactive oxygen species might actually mediate beneficial training adaptations as a part of a biologically useful signaling cascade that means that vitamin C could blunt the beneficial training adaptations that reactive oxygen species might induce such as increased mitochondrial number and function improved insulin sensitivity and glucose utilization and enhanced immune function among others
I'll bring up some concerns about whether vitamin C can blunt the effects of exercise especially in terms of mitochondrial biogenesis and function improved insulin sensitivity glucose utilization and enhanced immune function
some studies show that when vitamin C is taken with other supplemental antioxidants it might attenuate these beneficial effects