Paul Saladino· MD
there's the uncracked to 20 times risk corrected for age sex and comorbidity which is quite unfair to correct for comorbidity because it's kind of collinear it's connected but anyway 10x
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
there's the uncracked to 20 times risk corrected for age sex and comorbidity which is quite unfair to correct for comorbidity because it's kind of collinear it's connected but anyway 10x
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there's data now with vitamin D in India Indonesia the USA and the Philippines that show pretty much the exact same thing which is that there's about a 10x risk of severe coab in nineteen with a vitamin D level less than thirty nanograms per ml
the recent studies where vitamin D status is rough-and-tough after correction age and comorbidity so after correction for age around ten times more likely a severe outcome or death below twenty nanogram
specifically greater than 30 nanograms per deciliter in the blood the the rate of kovat severe outcomes is 10 times less
Severe vitamin D deficiency in people with COVID-19 was associated with a significantly higher mortality risk than COVID-19 patients with normal vitamin D levels (retrospective study in Bari, Italy).
for each standard deviation increase in serum vitamin D people were 7.94 times more likely to have a mild rather than severe COVID-19 outcome and were 19.61 times more likely to have a mild rather than critical outcome.
Data from 10 countries found people with vitamin D deficiency were twice as likely to experience severe COVID-19 complications, including death.
So they showed that there was a difference in mortality, that there was a difference in which ones went on to need ventilators.