The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
you can take amaloid beta 42 inject it into a worm muscle tissue and then activate heat shock proteins and it like prevents the aggregation and it prevents the the muscle sort of paralysis that occurs in these worms. Animal studies have been done looking at amaloid beta and heat proteins and Alzheimer's disease. Again, it's having a protective effect.
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Animal studies have been done looking at amaloid beta and heat shock proteins and Alzheimer's disease. Again it's having a protective effect.
has long been of interest to the aging community for its role in preventing protein aggregates, a hallmark of aging biology and neurodegenerative disease.
So there have been studies that have shown for example in animal models if you expose them to amaloid beta 42 the toxic protein that aggregates and associated with the pathology of Alzheimer's disease and you give these mice you know heat you make them express a lot of heat proteins it actually protects against the aggregation of this amaloid beta 42 too.