Andrew Huberman· PhD
now today we realize that there's a clear stratification of risk when it comes to Alzheimer's disease that tracks with those isoforms
Loading…
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
now today we realize that there's a clear stratification of risk when it comes to Alzheimer's disease that tracks with those isoforms
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's really a cardiovascular risk gene just as much as it is in alzheimer's risk gene
the fourth isoform of that is the high-risk one
we talk about why the protein that is coded by the apoe for isoform produces a much greater increase in the risk of Alzheimer's disease and cardiovascular disease