The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
your hypothesis is that the bacteria are making the lactate and they're disproportionately making the uh the less desirable form of it and that the L lactate that which you're actually measuring is probably not causing any of the problems associated with the sepsis it's just the it's you know it's telling you that something else is going on
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
because the microbes uh are producing reic lactate they're producing L lactate and D lactate and most of our body runs on the the form of lactate it's identified as L lactate but I think in there's a lot of deact going on that is formed in the lower bow