Andrew Huberman· PhD
There are a lot of animal data suggesting it can accelerate wound healing largely through increasing angiogenesis.
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
There are a lot of animal data suggesting it can accelerate wound healing largely through increasing angiogenesis.
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well it's been shown to increase blood flow to a given area by virtue of increased angiogenesis so basically to promote the development of new blood vessels to the entire injury site
bpc-157 somehow is able to recognize injured blood vessels and injured capillaries and then to promote the activity of a given enzyme called Enos or endothelial nitric oxide synthese which then causes more blood vasculature to form at the injury site and around the injury site
we've got bpc 157 which you can just basically frame up in your mind as promoting angiogenesis and wound repair through a variety of mechanisms but mainly the addition of new vasculature to the wound site
one of the clear effects of bpc-157 is to both encourage cellular turnover as well as cellular migration so new cells and cells moving into a given area as well as new blood supply through the promotion of this process we call angiogenesis
bpc-157 somehow is able to recognize injured blood vessels and injured capillaries and then to promote the activity of a given enzyme called Enos or endothelial nitric oxide synthese which then causes more blood vasculature to form at the injury site and around the injury site
we know from animal models that pc-1 157 increases angiogenesis the growth of capillaries in blood vessels we know this it can accelerate wound healing by virtue of increasing fibr blast motility
I also know that vascularization due to bpc157 even if it's injected locally into a given tissue is likely to occur globally throughout the body