Paul Saladino· MD
when they get to these steps that are required to initiate cardiovascular disease the very first step is the oxidation of linoleic acid in LDL which converts it from non-athrogenic LDL to atherogenic oxidized LDL
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
when they get to these steps that are required to initiate cardiovascular disease the very first step is the oxidation of linoleic acid in LDL which converts it from non-athrogenic LDL to atherogenic oxidized LDL
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the amount of seed oils you consume predisposes your or LDL to oxidation which is the required step to initiate atherosclerosis
and with Tucker and Jeff I talked a lot about cardiovascular disease and LDL and its propensity to oxidize when you're eating more linoleic acid
there are multiple studies which show that the more linolic acid you eat in seed oils the more linolic acid ends up in your LDL particles in your cholesterol in your vldl particles from the liver and that makes those particles more susceptible to oxidation oxidized LDL and LP little a are highly strongly associated with cardiovascular disease potentially causal in cardiovascular disease
potentially increased cardiovascular risk with increased oxidation of LDL LP little a and LPA 2
there are multiple studies in humans showing that when you consume seed oils your LDL is more likely to oxidize