The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
there's on the flip side there's familial dunnigan hyperlipidystrophy where people have normal levels of ldl but they have accelerated atherosclerosis and in the case of familial dunning and hyperlipidostrophy hyperlipidustrophy like that one is fascinating because there's a mutation that causes them to be insulin resistant
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
this dunnigan lipodystrophy they have insulin resistance but not elevated ldl guess what they get massive atherosclerosis it's very aggressive