Paul Saladino· MD
I think there is no good evidence that apob directly injures the endothelium if it did why why is that same amount of apob that is circulating in the human body not injuring our veins
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
I think there is no good evidence that apob directly injures the endothelium if it did why why is that same amount of apob that is circulating in the human body not injuring our veins
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
if that is the case why do we not see atherosclerosis that is the formation of atheromas and plaques in the veins of our body unless they are grafted into the arterial circulation but in a native human we do not see atherosclerosis in veins we only see atherosclerosis in arteries why is that because something else is required for the beginning of aoma