Paul Saladino· MD
when you see kids with a beta lipopular nibia and they don't have very much LDL they don't have much atherosclerosis
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
when you see kids with a beta lipopular nibia and they don't have very much LDL they don't have much atherosclerosis
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we have natural experiments right we can look at the a beta hyperlipoproteinemia patients who can't traffic cholesterol therefore they would be entirely dependent on their own cellular and dajin as production and they seem completely fine so that's not proof but we don't have proof to your point but it's certainly evidence to suggest