Paul Saladino· MD
perhaps ldl is a repair molecule perhaps ldl is getting pulled in by macrophages that are activated subendothelial entema is getting expansive because of ischemia we don't know exactly why that happens
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
perhaps ldl is a repair molecule perhaps ldl is getting pulled in by macrophages that are activated subendothelial entema is getting expansive because of ischemia we don't know exactly why that happens
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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I believe that LDL is part of the healing process in those arteries and perhaps LDL is moved into the sub-endothelial space in an active way but that I really remain unconvinced and I've talked about this at length on previous podcasts about that LDL and apob are directly atherogenic