We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
I think that what we will find my strong prediction I'll put money on it in 30 years if we ever do the study is that you know we'll find that these are divergent phenotypes without the flam authority markers without the underlying issue that you're talking about that it's causing you know small dense LDL that is causing the populations of HDL and triglycerides to become disordered I don't believe that this this LDL phenomenon is the best metric of atherosclerosis
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.