The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
and then you want to do a full smattering of of markers of inflammation so HSC RP phospholipase a2 what my local ops that I fell DL is a very important and there's other ones you can do do I surprise games there's there's you know I il-6 I don't one a you know different interleukins you can check for and I think HS ERP phospholipase a2 oxidized LDL I think those are really good markers for you to look at
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if you see a patient with an elevated c-reactive protein should you be concerned about it I mean yes probably but the question is it cardiac specific or not you can't really tell so that's why looking at fibrinogen and c-reactive protein and homocysteine and LP PLA 2 and oxidized LDL help you get a better picture of if there's inflammation
on the inflammation side we can see specific and nonspecific markers of inflammation so on the nonspecific side we can see things like fibrinogen c-reactive protein on the specific side you can see things like ox LDL LP PLA - ox phospholipid