The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when in during an inflamed State your body makes more VD vdl because it binds up what's called endotoxin which is yeah and it's a beneficial uh thing for you at that point it's kind of like a sponge soaking up this bad stuff that can do all this damage
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
You know, endotoxin release from the gut, one, it causes more VLDL production because VLDL sucks soaks it up. So that's part of the reason why inflammation is also correlated with an increase in LDL number. It also binds to ApoB, it binds to the part, it binds to where the LDL receptors bind so that LDL can't be recycled as well.