The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
we looked through epigenetic analysis I wi is the Royal Wii right is more Alex and Carolyn they look at the genes that were change that their activity change because of this cortisol exposure and through epigenetic analysis which is the space you know between DNA and RNA and there's like methylation patterns and all that and some gen activity changes some turn on some turn off
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
well why would happen if we expose some of organoids to cortisol
the first thing that they needed to do was identified that these neurons actually had these glucocorticoid receptors and that they were active and they did have them and they were active
interestingly the majority of the genes that we found there were genes that have been addressed in the literature as potentially being related to post trumatic stress disorder things like the glucocorticoid receptor genes and things that you would think of but there was another subset of genes that we identify that were novel and I was very interested in those because of my interest in accelerated aging because of stress and those were genes that are related to collagen formation