Rhonda Patrick· PhD
So if we can use CRISPR technology to go in and replace say, give it more anti-inflammatory capabilities, and this has been shown also in mice. I don't know if you've seen this study, but NF-kB, which is a gene that produces a protein that regulates a lot other genes that are pro-inflammation, so they cause inflammation, but it also has an anti-inflammatory component to it. And when you take away that anti-inflammatory component and put it in mice, what happens is every time there's an immune response, every time inflammation happens, which leads to chronic damage, as you talk about, there's a low level inflammation and it drives aging prematurely in mice.