The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
basically those drugs work by preventing the gobbling up of serotonin or re-uptake of serotonin into neurons after it's been released which leads to more serotonin overall which means to elevate serotonin. And indeed those drugs were and can be very useful for certain people to feel better in cases of depression and some other clinical disorders as well.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
The SSRIs worked by distinct mechanisms from the tricyclic antidepressants and the MAO inhibitors. As their name suggests, SSRI, Selective Serotonin Re-Uptake Inhibitors prevent serotonin from being wiped up from the synapse after two neurons talk to one another.
Selective serotonin reuptake inhibitor means, when a person takes this drug, some of those drugs include things like Prozac or Zoloft, the more typical or generic names are things like Fluoxetine. When people take those, more serotonin hangs out in the synapse and is able to be taken up by the neuron on the opposite side because of this selective reuptake inhibition.