The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Good example of this would be the drug Welbutrin also called bupropion which increases dopamine and norepinephrine. Wellbutrin bupropion was developed as an alternative treatment for depression because some people who take the so-called SSRI, selective serotonin reuptake inhibitors which as the name suggests increased serotonin suffer from serotonin related side effects.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
And it's no coincidence that Bupropion is also an antidepressant. It's a common antidepressant for people that experience negative side effects with the so-called SSRIs, the selective serotonin reuptake inhibitors that prevent them from taking those things like lessen libido or appetite, or in some cases increased appetite or any number of other side effects that some people, not all, but some people experience with SSRIs.
Maybe they'll take them off an SSRI entirely, give them a different type of antidepressant that works on a different set of neuromodulators, like dopamine and norepinephrine. So Wellbutrin would be a non-SSRI antidepressant.