Paul Saladino· MD
excessive gluconeogenesis causes hepatic insulin resistance Paradox and it's sequela the sequela which is a fancy word for the downstream effects are non-alcoholic fatty liver disease
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
excessive gluconeogenesis causes hepatic insulin resistance Paradox and it's sequela the sequela which is a fancy word for the downstream effects are non-alcoholic fatty liver disease
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What's going to happen is you're going to have increased hepatic glucose output because now your insulin resistant at the level of the liver so your liver can't inhibit the enzymes that cause gluconeogenesis