The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
So we reason if this is true and it's the gutb brain axis that's driving sugar preference then there should be a group of neurons in the brain that are responding to postingestive sugar and lo and behold we identify a group of neurons in the brain that does this and these neurons receive their input directly from the gut brain axis.
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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A key element of this circuit is that the sensors in the gut that recognize the sugar do not recognize artificial sweeteners. It's a completely different molecule that only recognizes the glucose molecule, not artificial sweeteners.
So these are gut cells that recognize the sugar molecule. I see. >> Send a signal and that signal is received by the veagal neuron directly. Got it? And this sends a signal through the gutb brain axis to the cell bodies of these neurons in the veagal ganglia and from there to the brain stem to now trigger the preference for sugar.