The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
we also we and others have also done work to show it can block satiety signals so we know endoc canabo at least um are capable of overriding leptin so leptin is an anorectic molecule comes out from the fat and usually we release it
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so leptin is an anorectic molecule comes out from the fat and usually we release it when we've eaten a lot and it's one of these things that tells our brain stop eating you know it works through again uh populations in the arcu nucleus and changes the way those neurons function to drive food seeking behavior um and we and others have shown previously that that you know if you elevate endocannabinoid you can override that
and so as you lose that endoc canabo function the animal is less interested in eating and so you can prevent these anorectic effects of leptin by like goosing up endoc canabo activity
endocannabinoids are these chemicals that were originally discovered in marijuana hence the name cannabis and endocannabinoid is a cannabis-like molecule that's made in your body um everybody is heard at least of the effect called the munchies where you um get stoned and it makes you want to eat and from what I've been told you don't crave steak you crave junk food right